Activating Stomach Satiety = Shrinking Waistline = Shrinking Risk of Early Death
Prof. Dr. Mohamed Elhashemy
November 15, 2008
Researchers from Imperial College London and the German Institute of Human Nutrition performed large study comparing the waist sizes of obese and normal persons with their body mass index. They found that excess fat around the waistline increases the risk of early death, even in people considered to be of normal weight.
The study published in The New England Journal of Medicine found that an increase in waist circumference of about two inches raised the risk of death in average by 15%.
I am working to develop a new technique to fight obesity through establishing an active satiating stomach spot using Luqaimat of peanuts that may help obese people to lower their intake of food volume down to 50% of their original food intake.
In 2008, I developed a breakthrough scientific theory that may delete most hunger bangs, and prevent gastric expansion, leading to loss of most of the obese people extra weight.
I published this theory under the name Elhashemy's Stomach Satiety Spot "ESSS". ESSS is the first theory to postulate that there must be a spot in the stomach that when being hit in a certain way will lead to non-expansion of the stomach musculature, hence less accommodation for food and as a result to lower caloric intake leading to progressive weight loss.
My theory does not come from vacuum; it is built on important scientific and clinical observations. I observed that many organs in our bodies have an anatomical and physiological spots which either initiate or delete certain function. An example of this is the blind and yellow spots of the retina, and the pacemaker of the heart, etc. I postulated that we may have got a spot in the stomach when pressing on it by solid food may result in activation of satiety sensation much more than usual.
The stomach mucosa is richly innervated by afferent vagal fibers which are mostly condensed at the fundus. The intramuscular afferents are gastric tension receptors. The function of the highly arborizing intramyenteric vagal afferent endings is not known up till now; I think that they are responsible for satiety sensations, which leads to blocking the gastric relaxation, hence reducing a person's ability and desire to gorge on.
I think that feeling of hunger is either due to lack of activation of brain satiety area namely the left amygdale, or lack of activation of the stomach satiety area namely Elhashemy's Stomach Satiety Spot "ESSS".
Nowadays, I am studying the effects of activation of ESSS on 20 of my morbidly obese patients who are under standard Luqaimat diet in comparison to 40 patients of similar BMI under same diet but without activation of such spot. After passage of 5 weeks I found a great difference between both groups amounting to 20% more weight loss in favor of first group. Both techniques gave breakthrough weight loss results.
The study published in The New England Journal of Medicine found that an increase in waist circumference of about two inches raised the risk of death in average by 15%.
I am working to develop a new technique to fight obesity through establishing an active satiating stomach spot using Luqaimat of peanuts that may help obese people to lower their intake of food volume down to 50% of their original food intake.
In 2008, I developed a breakthrough scientific theory that may delete most hunger bangs, and prevent gastric expansion, leading to loss of most of the obese people extra weight.
I published this theory under the name Elhashemy's Stomach Satiety Spot "ESSS". ESSS is the first theory to postulate that there must be a spot in the stomach that when being hit in a certain way will lead to non-expansion of the stomach musculature, hence less accommodation for food and as a result to lower caloric intake leading to progressive weight loss.
My theory does not come from vacuum; it is built on important scientific and clinical observations. I observed that many organs in our bodies have an anatomical and physiological spots which either initiate or delete certain function. An example of this is the blind and yellow spots of the retina, and the pacemaker of the heart, etc. I postulated that we may have got a spot in the stomach when pressing on it by solid food may result in activation of satiety sensation much more than usual.
The stomach mucosa is richly innervated by afferent vagal fibers which are mostly condensed at the fundus. The intramuscular afferents are gastric tension receptors. The function of the highly arborizing intramyenteric vagal afferent endings is not known up till now; I think that they are responsible for satiety sensations, which leads to blocking the gastric relaxation, hence reducing a person's ability and desire to gorge on.
I think that feeling of hunger is either due to lack of activation of brain satiety area namely the left amygdale, or lack of activation of the stomach satiety area namely Elhashemy's Stomach Satiety Spot "ESSS".
Nowadays, I am studying the effects of activation of ESSS on 20 of my morbidly obese patients who are under standard Luqaimat diet in comparison to 40 patients of similar BMI under same diet but without activation of such spot. After passage of 5 weeks I found a great difference between both groups amounting to 20% more weight loss in favor of first group. Both techniques gave breakthrough weight loss results.
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